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N-terminally truncated isoforms (N) lack the transactivation domain and appear to act in a dominant-negative manner by either competing with the TA isoforms of p63 and p53 for DNA binding sites or by direct protein interaction

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Leferink, P

Hepatitis C virus and vaccine development

IL-6 gene expression is activated by the N protein which binds to the NF-kB regulatory element on IL-6 promoter and facilitates its translocation from cytosol to nucleus

Correlating downregulated DEGs by RNA-seq and genes with decreased H3K4me3 ChIP-seq enrichment at the promoter showed an overlap of 51 genes, which are involved in several oncogenic pathways such as RAS and FGFR4 signalling (Fig