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These therapeutic options encompass RAS blockade, SGLT2 inhibition, GLP-1 receptor activation, endothelin-1 receptor blockade (atrasentan), vasopressin V2 receptor antagonism (tolvaptan), and selective mineralocorticoid receptor antagonism (finerenone), all demonstrating differential efficacy in retarding renal function decline (Li et al., 2024)

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It effectively prevents oxidative lipid damage and cell death in disease models, including Huntington's disease, periventricular leukomalacia, renal dysfunction, cerebral hemorrhage and cardiomyopathy (103,112,113)