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Venter C, Meyer RW, Nwaru BI, Roduit C, Untersmayr E, Adel-Patient K, Agache I, Agostoni C, Akdis CA, Bischoff SC, du Toit G, Feeney M, Frei R, Garn H, Greenhawt M, Hoffmann-Sommergruber K, Lunjani N, Maslin K, Mills C, Muraro A, Pali-Schll I, Poulson LK, Reese I, Renz H, Roberts GC, Smith P, Smolinska S, Sokolowska M, Stanton C, Vlieg-Boerstra B, OMahony L

In conclusion, we provide some evidence that TNF- can ultimately inhibit EBV reactivation by affecting GPX protein expression and thereby influencing potential ferroptosis status and ROS

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Additionally, without glutathione, oxidative stress activates the opening of mitochondrial permeability transition pores that results in the destruction of the membrane potential and halts ATP synthesis[21,30]

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eIF2, Eukaryotic translation initiation factor 2