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Such studies have either only characterized the metabolism of untreated resistant cells compared to sensitive cells [12, 15, 17], or also considered the effects of exposure to cDDP [13, 14], oxaliplatin [13, 16] or carboplatin [13] on both cell lines (sensitive and resistant)

However, a critical question arises: is Complex I deficiency a universal primary event in PD, or a secondary phenomenon under specific etiologies (e.g., environmental toxin exposure, certain gene mutations)

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Dunbabin V, Rengel Z, Diggle AJ (2004) Simulating form and function of root systems: efficiency of nitrate uptake is dependent on root system architecture and the spatial and temporal variability of nitrate supply

Kidney injury molecule-1 is elevated in nephropathy and mediates macrophage activation via the Mapk signalling pathway