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These processes enhance synaptic plasticity within corticolimbic circuits (hippocampus, medial amygdala, prefrontal cortex)

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Rucker R, Chowanadisai W, Nakano M

J Clin Sleep Med 2011

This abnormal mechanism promotes malignant transformation through dual effects: on the one hand, it simultaneously inhibits key tumor suppressor genes, and on the other hand, it activates oncogenic signaling pathways, ultimately leading to genomic instability