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6 In addition to reduction in pain intensity and attack frequency, there is also evidence supporting an increase in quality-of-life metrics after onabotulinumtoxinA injections for TN relative to placebo

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At low level of oxidative stress, antioxidants can restore cellular redox homeostasis

however, the continuous accumulation of senescent cells, through the secretion of pro-inflammatory molecules known as the senescence-associated secretory phenotype (SASP), ultimately leads to chronic inflammation, tissue dysfunction, and age-related diseases.34 Senolytic therapies are designed to exploit the vulnerabilities of senescent cells, particularly their altered molecular mechanisms that heighten their susceptibility to particular stressors.37 Distinctive features include modifications in the expression of pro-survival proteins and activation of specific survival pathways

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