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This metabolic vulnerability makes ferroptosis not only a major driver of tissue damage, but also a promising therapeutic target in diseases characterized by oxidative stress or therapy resistance [4, 5]

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Under homeostatic conditions, Nrf2 is bound to Kelch-like ECH-associated protein 1 (Keap1) and targeted for degradation through ubiquitination (McMahon et al., 2006)

This is particularly important during pregnancy, as the body undergoes significant changes that can increase oxidative stress levels

Temporal Summation of the Thermal Grill Illusion Is Comparable to That Observed Following Noxious Heat

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