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In addition, the researchers found that HBV enhances the reciprocity between p62 and Keap1 through binding HBx to p62, forming a HBx-p62-Keap1 complex in the cytoplasm, prompting the dissociation of Keap1-Nrf2, which contributes to activation of Nrf2 and up-regulation of G6PD expression

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That conclusion does not require overstating the evidence