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This paradoxthat patients with higher baseline severity showed higher rates of MELD-3 improvementis compatible with several mechanisms, including (but not limited to) a possible therapeutic effect of glutathione in inflammation-rich states, control of the precipitating event by concurrent standard care, and statistical regression toward the mean

They are generated through the activity of glycoside hydrolase enzymes, such as -galactosidase, which utilize lactose as a substrate

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