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Furthermore, they characterized IGF1R5, an sdAb that specifically binds to IGF1Rs at the BBB, serving as a ligand to trigger RMT, effectively delivering therapeutic cargo across the barrier [143]

Our aim was to investigate the effect of UA on the metabolism of endogenous antioxidant glutathione by estimating UA induced changes in the expression of glutamate-cysteine ligase, glutathione peroxidase, glutathione reductase, and glutathione synthetase in J744A.1 mouse macrophage cell line

Additionally, elevated glucose contributes to dysfunction in coupling glycolysis to glucose oxidation, pentose phosphate pathway, and polyol pathway

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