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The most rigorous published study is a double-blind, matched-pairs, parallel-groups trial by Bes et al

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[257] shows that supplementation of humanized dystrophic mice ( mdx 4cv /mTR G2 mouse model with humanized telomere lengths) with an orally active slow-release H 2 S prodrug (SG1002) results in maintenance of the ejection fraction (ET) at a level similar to wild-type mice, indicating preserved cardiac function

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