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Covalent modifications involving thiol groups of cysteine residues of KEAP1 initiate the activation of the Nrf2-ARE pathway [113]

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Kinoshita S, Onuma S, Yamazaki N, Shibukawa Y, Ozono K, Michigami T, et al

In atherosclerosis, arterial regions with turbulent blood flow, such as branch points, are particularly prone to lesions due to low shear stress and endothelial damage

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The Fragment does not bind to liver receptors, maintaining baseline IGF-1 levels