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PMC - Body protective compound-157 enhances alkali-burn wound healing in vivo: Discusses proliferation, migration, and angiogenesis in HUVECs via ERK1/2 pathway

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Furthermore, we show that targeting mitochondrial metabolism, particularly by inhibiting Complex I, sensitizes CRC cells to 5FU, resulting in delayed tumour growth and prolonged survival in preclinical models

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3,4 Combined Research Applications Because BPC-157 and Thymosin Beta 4 are studied for distinct but related cell signaling mechanisms including vascular signaling and actin-related cytoskeletal dynamics researchers may study them together to examine multiple pathways within the same experimental model