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Arce-Lpez et al

Our mechanistic findings can provide a rational approach for developing cancer treatments through targeting the ASXL1-BRD4 interaction with small molecule inhibitors, because the cancer-specific truncation variants of ASXL1 but not full-length ASXL1 have previously been shown to appreciably interact with BRD4 and induce myeloid malignancies in animal models 6,26,39

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Therefore, it is a regulated but non-apoptotic form of cell death (Dixon et al., 2012)

For short-chain peptides, mucosal absorption can be meaningful