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Next, we demonstrated that replicating key aspects of the lung alveolar micro-environment specifically, the airliquid interface (ALI), breathing-like motions (stretch), and the inclusion of an immune component significantly enhances the sensitivity of the smoke-induced lung-on-chip model

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These rigid, misshapen cells are prone to rupture and obstruct capillaries, impairing blood flow

In parallel, immunosuppressive cells and cytokines within the TME can dampen the immune response, making it difficult to achieve the desired therapeutic outcome [142,143,144]