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In the acute phase, the neuroinflammatory response worsens BBB injury by degrading tight junctions and the extracellular matrix, promoting neuroglial proliferation, and activating cardiomyocytes, thereby further increasing its permeability

KPV offers a potentially different risk profile

The excessive accumulation of cholesterol within mitochondria also promotes lipotoxicity, a condition in which lipid metabolites induce OxE and inflammation

Transferrin receptor internalization sequence YXRF implicates a tight turn as the structural recognition motif for endocytosis

L-Carnitine is a naturally occurring compound that facilitates the transport of fatty acids into mitochondria for beta-oxidation

This is the case even in tropical countries like Brazil and Thailand