glutathione and parkinsons disease iv parkinson s Parkinson's disease: Is this the elephant in the room? Central nervous system uptake of Possible neuroprotective mechanism of DHA,

Mitophagy is closely linked to mitochondrial fission and fusion processes (220)

Kidney disease is serious and needs veterinary diagnosis

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The neurotoxic threat of micro-and nanoplastics: evidence from in vitro and in vivo models

Several interesting key findings of their study include: a) the cell death induction effect by curcumin was independently through p53 and caspase upregulation, which effectively dodge the atypical stimuli by both p53 and caspase that exert increased the survival and chemoprotection to tumor cells, b) the binding activities of AP-1 and NFB were decreased (where the constitutively over-expression of these genes is a marker in GBM), c) curcumin decreased the expression of genes which conferred chemoresistance such as IAP and Bcl family, and d) the expression of DNA repair enzymes (MGMT, Ku70, Ku80 and DNA-PK cs ) believed to be involved in chemosresistance and radioprotection was inhibited by curcumin

Excess glutamate or poor glutamate clearance can overstimulate brain circuits involved in error detection, intrusive thoughts, compulsive urges, anxiety, and insomnia