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These inflammatory mediators have been shown to modulate BBB permeability and activate microglia, leading to a sustained neuroinflammatory environment that increases neuronal vulnerability, and potentially facilitating peripheral syn aggregation and spread [152, 153]

Immune modulation with a staphylococcal preparation in fibromyalgia/chronic fatigue syndrome: relation between antibody levels and clinical improvement

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Using next-generation sequencing, a clinical study analyzed the B-cell receptor repertoire of the transitional Breg subpopulation with CD19 + CD24highCD38high phenotypes in patients with MS [48]

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