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For instance, USM [H]L vesicles integrate encapsulated enzymes to degrade uric acid and hydrogen peroxide, generate photothermal effects for inflammation disruption, repolarize M1 macrophages to M2 phenotypes, and reprogram metabolic/immunological pathways for gout resolution (Yang et al., 2020)

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Material design can also target cuproptosis-like pathways by addressing Gram-positive and Gram-negative differences

miR-346 functions as a pro-survival factor under ER stress by activating mitophagy

Developing KDML105 backcross introgression lines using marker-assisted selection for QTLs associated with drought tolerance in rice

As such, overexpression of ArfGAP1 in mammalian cells induces the redistribution of the entire Golgi complex to the ER via vesicular intermediates [39, 41, 42]