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However, in all cases, sustained UPR activation promotes neurodegeneration by exacerbating ER stress, initiating apoptotic pathways, and impairing cellular recovery mechanisms

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[DOI] [PMC free article] [PubMed] [Google Scholar] 317.Heurtier V, et al

During senescence and in the absence of pathology, microglia undergo fragmentation of the cytoplasm, loss of branching, and shortening of cell processes (Streit et al., 2004), leading to a decrease in the microglial arbor area, showing a dendritic, less symmetrical, more elongated, and less circular appearance of the cells (Tremblay et al., 2012

It is small, stable and extraordinarily bioactive: published research links GHK-Cu to modulation of more than 4,000 genes affecting collagen , elastin , antioxidant defence , angiogenesis and wound repair