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BH4 is regenerated from its oxidized form by dihydrofolate reductase (DHFR), ensuring its availability as a cofactor

However, once the protein sequences of representative members of these families were available, it was clear that they were closely related

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We thus hypothesized that CD38 upregulation might have a fundamental pathogenic role in SSc fibrosis, and further, restoring NAD + homeostasis will prevent the process

[DOI] [PMC free article] [PubMed] 1046.Dubrovska A, et al

150 In conclusion, nonhistone lactylation, as a dynamic regulatory mechanism, plays a critical role in various physiological and pathological processes within cells