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To mitigate the accumulation of harmful glycolytic byproducts such as advanced glycation end-products (AGEs), sorafenib-treated cells reroute excess dihydroxyacetone phosphate (DHAP) toward glycerol-3-phosphate (G3P) synthesis, supporting glycerolipid metabolism, NAD + regeneration, and redox balance, rather than producing D-lactate via the glyoxalase pathway

Both hesperidin and CoQ10 treatment for 21 days significantly restored the GSH level hippocampal region suggesting their protective defense action

These findings position mtNOS1 as an actionable target in overcoming therapy resistance

Biochimie 86 , 825831 (2004)

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