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Histology of the monkey anterior segments showed that treatment with the HepII domain disrupted the contractile properties of the TM/SC similar to what was observed in monkey eyes treated with the H-7 inhibitor (Sabanay et al., 2000)

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This results in increased production of desirable hormones (LH, GH), and decreased the production of undesirable ones (cortisol and somatostatin)

Neuroprotection of GluK1 kainate receptor agonist ATPA against ischemic neuronal injury through inhibiting GluK2 kainate receptor-JNK3 pathway via GABA(A) receptors

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