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T h 2, T h 17, T h 22, and T fh cells) remain controversial, there is a propensity of evidence to support a clear proatherogenic role for T h 1 cells and an atheroprotective role for Tregs

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Th1 cells that undergo ferroptosis may perish and emit damage-associated molecular patterns (DAMPs), which intensifies the inflammatory response

[187] suggested that artemisinin reverses suppressed Treg levels by inhibiting ERK phosphorylation in AR mice