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Since co-treatment of UK-5099 and JMF4073 depleted dTTP pool and suppressed the growth of T315-Bcr-Abl-32D cells, we proposed that reducing GSH biosynthesis induces not only oxidative stress but also replication stress in tumor cells, which demands the synthesis of dTTP and dCTP for survival

Thus, acute Ca 2+ flux and persistent Ca 2+ dyshomeostasis are distinct cell death phenomena with the latter primarily affecting biosynthesis of ferroptosis substrates

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This article reviews the clinical evidence for TRE, indicating its potential to facilitate weight reduction, improve insulin resistance, and ameliorate dyslipidemia

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