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With mitochondrial loss and injury, the RPE has reduced baseline oxygen consumption and reserve capacity, and elevated ROS, making them poorly responsive to stress 50

The first few weeks produce modest changes as the dose titrates up

doi: 10.1038/ncomms15637 119 DooleyHCRaziMPolsonHEGirardinSEWilsonMIToozeSA

In conclusion, the cardioprotective role of ALA against DOX cardiotoxicity was dependent on PDK4-mediated regulation of oxidative stress and mitochondria metabolism

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