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To determine whether SLC22A17 is required for LCN2-driven ferroptosis, we transiently silenced endogenous expression of Slc22a17 using siRNA (Fig

GA reduces myocardial hypertrophy induced by transforming growth factor beta 1 and dysfunction caused by transverse aortic constriction, while also decreasing deposition of type I collagen, thereby preventing cardiac fibrosis (Jin et al., 2018)

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Cathepsin G and its role in inflammation and autoimmune diseases

This is evidenced by its ability to reduce the number of iron-stained cells in the SN, reverse the loss of dopamine and TH-positive neurons, and its mechanism is linked to iron chelation-mediated reduction of oxidative stress (256)

van Durme CMPG, Wechalekar MD, Landew RBM