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Toxicology 105(23):267274 Jones DP, Carlson JL, Mody VC, Cai J, Lynn MJ, Sternberg P (2000) Redox state of glutathione in human plasma

Understanding the "Master" Molecules To understand why they work so well together, we first need to look at what they do individually

[DOI] [PubMed] [Google Scholar] 81.Hopkins RE, Dobbin M, Pilgrim JL

In addition, mitochondria lack more advanced repair pathways, such as nucleotide excision repair and homologous recombination, which makes mtDNA particularly susceptible to the accumulation of mutations [35], which leads to a higher rate of damaged mtDNA in mitochondria and further aggravates oocyte damage

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[] The interplay between mtMOF and the deacetylase SIRT3 dynamically regulates mitochondrial protein acetylation levels