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105 ), the immature innate immune system in neonates has a low capacity to generate ROS, so pro-oxidative processes in neonatal sepsis are limited to intracellular compartments of affected tissues

10.1371/journal.pone.0215277 PLoS One 37 CanoE.MahadevanL

Additionally, this increase in microglia numbers, which facilitated glial cell proliferation, occurred at a critical time during optic glioma development (91)

In a neonatal chronic hypoxia mouse model mimicking PBI, both compounds promote neural progenitor cell proliferation and oligodendroglial fate acquisition, with leucovorin further enhancing differentiation