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Glutathione, in its oxidized state no longer functions as an antioxidant

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These mechanisms include efficient recycling through the vitamin B12-dependent methionine synthase, which is the sole enzyme in the brain responsible for converting Hcy into methionine

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Conversely, ferroptosis-related release of oxidized byproducts and iron ions feeds back to escalate inflammation and further damage cartilage and synovium, establishing a vicious cycle

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