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The pathological hallmarks of AD include extracellular amyloid-beta (A) plaques and intracellular neurofibrillary tangles (NFTs) composed of hyperphosphorylated tau protein, which accumulate in the brain over decades before clinical symptoms manifest [3-4]

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For instance, we have shown that elderly patients with treated hypertension exhibit increased CuZn-SOD activity and elevated GSH levels (28), which may suggest that lowering blood pressure during antihypertensive treatment is at least in part due to a CuZn-SOD-GSH-dependent mechanism

Various curcumin formulations have been developed to address this limitation

Figure 1 Despite significant challenges in managing neurodegenerative diseases, advances in mechanistic understanding are rapidly emerging

Frey, A