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Description

Endoplasmic reticulum stress and mitochondrial cell death pathways mediate A53T mutant -synuclein-induced toxicity

10.1124/jpet.115.231456 72

This approach may provide an attractive alternative to directly target the desired oncogene (or protein of another pathogenic activity), focusing instead on developing tools to target the zDHHCs controlling the proper (or pathological) localization of the protein