slc25a39 is necessary for mitochondrial glutathione import in mammalian cells

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells DLAT sustains redox homeostasis and prevent colorectal cancer from ferroptosis by regulating SLC25A39-mediated transport SLC25A39 identified as a key

Name: slc25a39 is necessary for mitochondrial glutathione import in mammalian cells DLAT sustains redox homeostasis and prevent colorectal cancer from ferroptosis by regulating SLC25A39-mediated transport SLC25A39 identified as a key
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SLC25A39 identified as a key regulator of hepatocellular carcinoma progression through the mitochondrial ROScytochrome ccaspase signaling axis Cellular & Molecular Biology Letters Springer Nature Link SLC25A39 protein levels are directly regulated by cellular GSH Download Scientific Diagram Dual regulation of SLC25A39 by AFG3L2 and iron controls mitochondrial glutathione homeostasis ScienceDirect Glutathione driven redox decisions in cell survival and death Controlling glutathione entry into mitochondria: potential roles for SLC25A39 in health and (treatment of) disease Signal Transduction and Targeted Therapy SLC25A39 is necessary for mitochondrial glutathione import in mammalian cells bioRxiv

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222 GPX4 is one of the main antioxidant protective enzymes in pancreatic cells and plays an important role in reducing lipid peroxides and protecting cells from ferroptosis in T2DM