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Toxic effects are also associated with the inhibition of key enzymatic activities, including -amylase, peptidase, malate dehydrogenase, protease, and pyruvate kinase, all of which are essential for starch hydrolysis, energy metabolism, and protein mobilization

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[376] observed that despite the addition of quercetin and chlorogenic acid to a DSS-induced UC mice did not protect against indicators of injury and inflammation, or fecal SCFA concentrations compared to the control diet, these compounds influenced the expression of various injuries repair molecules, pro-inflammatory cytokines, SCFA transport proteins, and NF-B inhibitory molecules

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