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In erastin-resistant cancer cells (DU-145, CHL-1 and SK-Mel5), an increased expression of AKR1C (including AKR1C1, AKR1C2 and AKR1C3) prevents ferroptosis by reducing the end products of lipid peroxides (AA/AdA-PE-OOHs) to the corresponding nontoxic lipid-derived alcohols (AA/AdA-PE-OHs) 84 (Fig