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Most importantly, GATA-1 upregulates autophagy-related and cell cycle-regulating genes, pro-survival anti-apoptotic Bcl-2, and downregulates myeloid genes to ensure the maintenance of erythroid specification [44]

neither the cystinecysteine redox shuttle nor direct transport by an unidentified SLC25 member has been validated, leaving the sulfur-supply step a potential bottleneck for Fe-S assembly flux