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Louis, MO, USA) and 875 l of PBS per 110 6 cells

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Histopathologically, periportal to massive coagulative necrosis with vacuolar degeneration of hepatocytes was seen in the liver of the WT mice at 75 mg/kg (3 of 7 mice) and the Aldh2*2 KI homozygous mice at 50 mg/kg (1 of 3 mice) and 75 mg/kg (11 of 11 mice) at 24 h after AA exposure (Table 3 and Fig

These findings underscore the interplay between iron-dependent oxidative damage and metabolic dysregulation in driving diabetic cardiac pathology, highlighting ferroptosis as a potential therapeutic target for mitigating diabetes-associated cardiac dysfunction