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CAC seems to be a target of a first-in-class treatment for the precancerous skin condition actinic keratosis

(PubMed) Lykkesfeldt J, Christen S, Wallock LM, Chang HH, Jacob RA, Ames BN

Other components of the DNA damage and BER machinery have been identified as specific readers of 5fC or 5caC, including p53, TDG, PARP, GADD45 and NEIL1/2 (Spruijt et al., 2013), and depletion studies of these factors in cells have shown that their activity is required to prevent DNA hypermethylation (Li et al., 2015

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The conversation shifts from 'What unknown thing is causing this problem?' to 'How do we manage the known physiological responses to GH elevation?' That is a world of difference

ECs, ACs, and PCs all secrete ECM and form the BM