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The body possesses a highly developed antioxidant system that defends against ROS, wherein catalase, superoxide dismutase (SOD), and glutathione peroxidase are the key antioxidant enzymes [14]

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We investigated the efficacy and mechanism of ferroptosis inducers RSL3 and FIN56 on persister cells, which are glutathione peroxidase 4 inhibitors

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Mechanisms of synaptic dysfunction and excitotoxicity in Huntingtons disease

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Carnosine levels are progressively modulated at embryonic stages in the brain

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TBK1, therefore, appears to play an essential role in late autophagic flux

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