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2022;13:1664-302X

Subsequently, regardless of the mechanism involved, in mitochondrial DNA damage or in the direct involvement of prooxidant factors in cellular mechanisms, the cellular response to stress will produce an overexpression of proinflammatory genes with increasing levels of prooxidant factors (Liguori et al., 2018)

In agreement with these findings, we found that NQO1 overexpression under HFD favors an increase in the activities of CRAT and SIRT3 that buffers mitochondrial acetyl group availability while counteracting mitochondrial protein hyperacetylation in the muscle concomitantly with enhancement of glucose and NAD metabolism in liver, skeletal muscle and adipose tissues

Nevertheless, the precise mechanisms responsible for the selective paternal mitochondrial elimination, remain largely elusive

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5.2 Flavonols Flavonols, also known as 3-hydroxyflavones, are characterized by a 2-phenylchromen-4-one backbone with a hydroxyl group at the C3 position