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The activation of AMPK broadly suppresses proinflammatory signaling pathways, including the JAK-STAT, NF-B, C/EBP, CHOP, and HIF-1 pathways, which in turn inhibits the expansion and immunosuppressive function of myeloid-derived suppressor cells (MDSCs) 42

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These medications work by mimicking the action of incretin hormones, which are naturally released in response to food intake

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This combination is of particular interest in experimental models studying endocrine physiology, metabolism, muscle function, bone metabolism, tissue regeneration, and the interaction between GHRH signaling and ghrelin receptors