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Activation of an H 2 O 2 -generating NADH oxidase in human lung fibroblasts by transforming growth factor beta 1

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[6] This interaction is hypothesized to trigger a cascade of intracellular signaling mechanisms

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[22] Lung cancer cells stimulated using glucocorticoids were induced into a reversible dormancy state which was dependent on the IGF-1R and its accompanying survival signaling pathways