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A study found that the NRF2-KEAP1 complex keeps shuttling constantly between the cytoplasm and the nucleus, and in the nucleus, the nuclear protein prothymosin competes for NRF2 binding to KEAP1 and enables the liberated NRF2 to activate NRF2 target genes [15]

In the first part of this study, NTAPP increased the expression of genes related to the biological oxidation-mediated- and the Wnt/-catenin- pathways

To view a copy of this licence, visit About this article Cite this article Liu, M., Zang, X., Vlahakis, N.W

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