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ROS-related anticancer mechanisms include mitochondrial dysfunction, p53-dependent apoptosis, iron-dependent ferroptosis, activation of endoplasmic reticulum stress, inhibition of growth signaling pathways (such as the epidermal growth factor pathway, EGF), among others

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For example, ASCT2 facilitates the uptake of Gln into the cells, whereas LAT1 is responsible for transporting Gln out of the cell in exchange for leucine, thereby activating mTOR

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