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Given the strong association between steatotic liver disease and cardiometabolic disorders [10] , this research suggests the possibility of linking deranged copper metabolism with metabolic dysfunction-associated steatotic liver disease (MASLD)

Introduction Since its discovery in 1986 ( The NF-B signaling pathway NF-B is a family of transcription factors comprised of several members: RELA (p65/RelA), c-REL (c-Rel), RELB (RelB), NFKB1 (p105/p50), and NFKB2 (p100/p52) ( Figure 1A )

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Once -arrestin is bound to a GPCR, it undergoes a conformational change allowing it to serve as a scaffolding protein for an adaptor complex termed AP-2, which in turn recruits another protein called clathrin

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