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Specific instances of an effect of host Se deficiency on RNA viruses or associated diseases Coxsackie virus and Keshan disease In the 1990s, Beck and colleagues showed that Se deficiency in mice, with its associated low or absent activity of protective GPX1, caused mutations in a non-virulent stain of coxsackie virus B3 that lead to the development of a virulent strain that caused myocarditis, even in a subsequent mouse that was Se-sufficient (Reference Beck, Shi and Morris18)

Mol Aspects Med 30(12):7785 Park JW, Mieyal JJ, Rhee SG, Chock PB (2009) Deglutathionylation of 2-Cys peroxiredoxin is specifically catalyzed by sulfiredoxin

Studies have shown that these systems effectively maintain therapeutic drug levels in the eye, enhancing treatment adherence and reducing the incidence of ocular surface disease associated with conventional therapies (243, 244)

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Mutations in SELENBP1, encoding a novel human methanethiol oxidase, cause extraoral halitosis

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