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For this experiment, co-IP was first carried out to pull down TDE0362 cleaved products from the SCSs using polyAb and then probed against mAb-N7

For Tirzepatide users, MK-677 presents a trade-off

Under physiological conditions, it supports synaptic plasticity [346], whereas under pathological states, such as during astrocytic necroptosis, proBDNF may exert neurotoxic effects and promote apoptotic signaling [349]

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The foundational mechanism paper that established MOTS-c as a therapeutically relevant peptide is also worth citing directly: a peer-reviewed Cell Metabolism study by Lee and colleagues showing that the mitochondrial-derived peptide MOTS-c promotes metabolic homeostasis and reduces obesity and insulin resistance through the AMPK pathway

Compensatory glutamine metabolism promotes glioblastoma resistance to mTOR inhibitor treatment