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As mentioned previously, oxidative stress acts as a potent catalyst for releasing inflammatory cytokines, 287 whereas MT1/2 effectively inhibits the activation of pro-inflammatory cytokines like IL-6, IL-12, and TNF-

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Microbial metabolites and inflammatory mediators can alter vagal afferent transmission, influencing immune-regulating pathways in the brainstem and midbrain [108]

All of these factors can be alleviated by the cytoprotective and anti-inflammatory actions of IGF-1 [105, 123, 247249]