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At the biochemical level, GHK-Cu is understood to exert its effects through two converging mechanisms: direct modulation of copper-dependent enzymatic activity and transcription factor activation

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By scavenging ROS, antioxidants help mitigate oxidative damage and reduce inflammation, offering a protective mechanism [5,8,9]

By targeting both, medications like bimekizumab may offer greater suppression of inflammation, which may translate to improved clinical outcomes for some patients, compared with blocking IL-17A alone. Its about achieving a more complete blockage of the inflammatory signals behind psoriasis, says Lawrence Green, MD, a clinical professor of dermatology at the George Washington University School of Medicine in Washington, DC