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This paper provides a theoretical framework for understanding and potentially treating VaD-related neuronal injury through the investigation of ferroptosis mechanisms, the biological functions of HO-1, and its role in regulating ferroptosis

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In vivo and in vitro studies demonstrated that oxidative stress and inflammatory cytokines triggered by chronic inflammatory process result in accelerated atherosclerosis in vascular beds (9, 10)

Hexameric calgranulin c (S100A12) binds to the receptor for advanced glycated end products (RAGE) using symmetric hydrophobic target-binding patches

Smad, small mother against decapentaplegic